Congratulations on losing 50 lbs and all your hard work at the gym. Obviously you are doing something right.
Don't be discouraged because your cholesterol only dropped two points.
The vast majority of cholesterol is manufactured in the body, to meet the body's demands for cholesterol. Only the smaller portion, roughly 15% comes from diet.
In human physiology, there is a built-in feedback system which regulates how much cholesterol is produced. If a person restricts dietary cholesterol, the body will respond by making more cholesterol to meet current physical needs.
Muscles need cholesterol for cell growth as cholesterol is one of the building blocks to healthy cells.
The brain is the one organ most dependent upon cholesterol to function properly. Its so important, the brain even churns out its own cholesterol for proper neurological function and memory recall.
All our steroid hormones are made from cholesterol. Steroid hormones are required for muscle toning and fitness as well as libido.
Don't be disappointed. Nature is watching out for you.
For a long time statin drugs used the disclaimer "Has not been shown to prevent heart disease, heart attacks or strokes" Then about a year or so ago, they dropped the disclaimer. I don't know if that is what JJTopp99 was referring to or not.
Its true that the current literature does say LIPITOR can lower risk in certain individuals, BUT its important to understand by how much, and just who is making that claim.
Gaining an understanding of the difference between relative risk reduction and absolute risk reduction is way to gain a clearer picture.
A Relative risk reduction could be as high as 40% reduction, while the corresponding absolute risk reduction could be as low as 1%, even less in some cases.
Then it also comes down to individual acceptance of drug company sponsored study results. Some people accept it, others don't claiming bias study results. Everyone has differing attitudes towards this.
Not all studies are Drug industry funded, but many cholesterol lowering drug studies were.
I hate to say it, but it all boils down to what we believe. At this point in time we still don't have a definitive underlying cause for atherosclerosis. We have risk factors identified, but the underlying mechanism has yet to be determined.
On a positive note, hopefully in out lifetime we will all get to see the answer.
"But there is no proof that if it lowers heart attacks."
That is the ultimate outcome. Does something prevent heart attacks and save lives?
Its a tough question because its so difficult to measure. When comparing two groups, we can compare the number of heart attacks in each group, but we really don't know who was destined to have a heart attack and who wasn't. Its difficult to measure how any treatment may have avoided an event in any individual.
Bill, they say statins stabilize plaque. Where does that data come from? What do they mean by stabilize?