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New US Diet Guidelines: What Not to Eat
Instead of just giving Americans a list of healthy food choices, the federal government takes a stab at showing us what not to eat. Get the facts here.
After eight years, the researchers found that the risk of death from prostate cancer among statin users was one per cent compared with five per cent for non-users
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Click the link above for information on what the results of a lipid panel can mean. As always, please discuss your results with your doctor.
HaylenView Thread
HaylenView Thread
Click the link above to learn more.
If you're currently taking a statin or have been on one in the past, did you notice this side effect?
HaylenView Thread
What do you think has contributed to the decline?
HaylenView Thread
Click the link above to read Dr. Richman's latest post about statin side effects.
Are you on a statin? Are you having side effects or are you happy with the cholesterol lowering benefits?
HaylenView Thread
I have found my voice and want to share my experiences with others who have been diagnosed with heart disease and other chronic diseases. My goal is to provide inspiration and a plan to follow to assist you.
Why am I writing this blog?
I want to share a little secret. It is very simple and the skeptics among us will doubt its effectiveness based on its simplicity.
The Secret:
Eat Plants. Lots of them.
My personal issue began with a very gradual increase in body weight from 200 to a high of 235 pds. My blood pressure was consistently increasing as well. I did not suffer a heart attack but had all the classic signs of angina. I noticed most of the symptoms during my frequent exercise. I could not run more than a quarter of a mile without walking. I was not out of breath, I just could not go further. Another very telling sign was the increase in my neck size of a three to four year period. I frequently would get flushing in my face, particularly when I was stressed.View Thread
Brand name and generic versions of statin drugs must carry these warnings on their labels, the FDA today announced.
Click the link for more information as well as specific medication brand names and types.
Have you experienced any adverse side effects? If so, how are you addressing?
HaylenView Thread
Cholesterol-Lowering Statin Drugs: Fequently Asked Questions with Steven Nissen, MD, chair of cardiovascular medicine at the Cleveland Clinic.
Do you agree or disagree with Dr. Nissen?
HaylenView Thread
Brand name and generic versions of statin drugs must carry these warnings on their labels, the FDA today announced. Click the link above for a list of the brand name and generic meds that must now carry the warning.View Thread
(Note: Don't trade a prescription for statin pills for a flu shot or other protections just yet. Researchers say their findings need to be confirmed in a larger clinical trial.)
HaylenView Thread
Are you taking the risks of high cholesterol seriously?
How do you stay on track to keep your cholesterol in check? Any tips or tricks for those looking for answers here?
HaylenView Thread
Click here for tips from the WebMD Cholesterol Management Health Center that gives tips: 10 Ways to Boost Your Good Cholesterol
Included are drinking three cups of orange juice a day, substitution soy for animal-based products, aerobic exercise and losing weight.
What do you do to boost your HDL?
HaylenView Thread
For me, information is easier to process when it comes with photos. (I even start to space out with my daughters 1st grade math so I found this helpful.)
Do you know your numbers? I have scheduled my fasting blood test for next Tuesday and will report back when I get results!
HaylenView Thread
A total of 3,277 healthy men without chronic diseases at baseline were included in the analyses. The median total cholesterol concentration at baseline was 6.5 mmol/L (251 mg/dl) (interquartile range 5.8 to 7.3 mmol/L, 224 to 282 mg/dl) and, in 2000, was 5.2 mmol/L (201 mg/dl) (interquartile range 4.6 to 5.9 mmol/L, 178 to 228 mg/dl).
During the follow-up period, 1,773 men (54%) died. A strong and graded relation was found between the cholesterol level and total mortality, with the men with a cholesterol level ≤4 mmol/L (154 mg/dl) having the lowest mortality. In all, the men with the lowest cholesterol gained the most life years. However, no association was found with the cholesterol level in 2000 (when 16% were using statins) and subsequent mortality.
The lowest (≤4 mmol/L) cholesterol value in midlife also predicted a higher score in the physical functioning scale of RAND-36 in old age. In conclusion, a low total cholesterol value in midlife predicts both better survival and better physical functioning in old age.View Thread
Click the link to answer questions and get your report card!
HaylenView Thread
Study Shows Hot Flashes in Menopause May Predict High Cholesterol
After taking into account other heart disease risk factors they found hot flashes and, to a lesser extent, night sweats to be predictive of higher cholesterol The more hot flashes the women had, the higher their LDL "bad" and HDL "good" cholesterol.
Haylen
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The study was published online August 28, 2011 in the European Heart Journal to coincide with the ESC presentation.
ASCOT-LLA and the long-term effects of atorvastatin
The results of ASCOT-LLA were first presented and simultaneously published online in the Lancet in 2003 [2 >. As reported by heartwire , lipid lowering with atorvastatin resulted in a significant 36% reduction in the primary end point of fatal coronary heart disease and nonfatal MI after a median follow-up of 3.3 years. At the time the study was stopped, there was a nonsignificant trend toward reduction in all-cause mortality. Upon completion of ASCOT-LLA, investigators continued to collect mortality data and evaluated the mortality outcomes in participants originally randomized to atorvastatin or placebo in the ASCOT-LLA arm for a median of 11 years.
At the end of the extended follow-up, all-cause mortality was significantly reduced by 14% (hazard ratio [HR> 0.86; 95% CI 0.76-0.98), and noncardiovascular mortality was significantly reduced by 15% (HR 0.85; 95% CI 0.73-0.99). There was no difference in death from cardiovascular causes.
Looking more closely at deaths from noncardiovascular causes, investigators found that deaths due to cancer were not statistically significant between those treated with atorvastatin vs placebo. There was, however, a significant 36% reduction in deaths due to infection and respiratory illness (HR 0.64; 95% CI 0.42-0.97), driven primarily by deaths due to infection.
During the session, Sever noted there are emerging data on the effects of statins on infection, with preclinical studies showing statins modulate neutrophil function, reduce proinflammatory cytokine release, improve vascular function, have antithrombotic properties, and improve outcomes from pneumonia and sepsis. Results of other observational studies have suggested that prior statin use also reduces mortality from sepsis. Despite these observations, Sever said that there is still the possibility of confounding bias in some of the observational studies that have shown a benefit of statins in pneumonia and sepsis and that caution should be used when interpreting such results until a randomized clinical trial is performed.View Thread
The study was published online August 28, 2011 in the European Heart Journal to coincide with the ESC presentation.
ASCOT-LLA and the long-term effects of atorvastatin
The results of ASCOT-LLA were first presented and simultaneously published online in the Lancet in 2003 [2 >. As reported by heartwire , lipid lowering with atorvastatin resulted in a significant 36% reduction in the primary end point of fatal coronary heart disease and nonfatal MI after a median follow-up of 3.3 years. At the time the study was stopped, there was a nonsignificant trend toward reduction in all-cause mortality. Upon completion of ASCOT-LLA, investigators continued to collect mortality data and evaluated the mortality outcomes in participants originally randomized to atorvastatin or placebo in the ASCOT-LLA arm for a median of 11 years.
At the end of the extended follow-up, all-cause mortality was significantly reduced by 14% (hazard ratio [HR> 0.86; 95% CI 0.76-0.98), and noncardiovascular mortality was significantly reduced by 15% (HR 0.85; 95% CI 0.73-0.99). There was no difference in death from cardiovascular causes.
Looking more closely at deaths from noncardiovascular causes, investigators found that deaths due to cancer were not statistically significant between those treated with atorvastatin vs placebo. There was, however, a significant 36% reduction in deaths due to infection and respiratory illness (HR 0.64; 95% CI 0.42-0.97), driven primarily by deaths due to infection.
During the session, Sever noted there are emerging data on the effects of statins on infection, with preclinical studies showing statins modulate neutrophil function, reduce proinflammatory cytokine release, improve vascular function, have antithrombotic properties, and improve outcomes from pneumonia and sepsis. Results of other observational studies have suggested that prior statin use also reduces mortality from sepsis. Despite these observations, Sever said that there is still the possibility of confounding bias in some of the observational studies that have shown a benefit of statins in pneumonia and sepsis and that caution should be used when interpreting such results until a randomized clinical trial is performed.View Thread
HaylenView Thread
Recommended by the National Cholesterol Education Program of the U.S. National Institutes of Health, the diet calls for limiting fats in your diet.
Has anyone tried?
HaylenView Thread
I'm great with exercise - not so much with food....
HaylenView Thread
What do you do to keep from sitting for a long period of time?
HaylenView Thread
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